Being inactive for as little as a few days makes muscles weaker and smaller, but that is not all you lose. Two new studies show that just two weeks of decreased physical activity brings you closer to becoming diabetic by decreasing your body's response to insulin, raising blood sugar levels after meals and making you fatter.
Today, more than 29 million people in North America are diagnosed as being diabetic, another 86 million have pre-diabetes, and most diabetics have not even been diagnosed. More than 88 percent of North American adults have their blood sugar levels rise too high after they eat.
Some foods raise blood sugar far more than others, and a high rise in blood sugar after meals can increase risk for diabetes, heart attacks and premature death. A study from Israel shows that some people develop surprisingly high blood sugar levels after eating foods such as bread, pizza, potato, tomatoes or bananas, while others do not develop the expected rise in blood sugar even after drinking a sugared soft drink or eating a cookie.
Type II diabetes means that a person is diabetic because his cells do not respond to insulin. Recent research shows type II diabetes is linked to gut bacteria that invade the inner lining of the colon, while the dominant bacteria of most non-diabetics do not try to invade the inner lining of their colon.
North America's epidemic of obesity, diabetes and heart attacks appears to be fueled by storing fat in the liver, muscles and around organs, rather than just in fat cells, excess calories, sugar added to foods and drinks and lack of exercise.
Diabetes can be caused by repeatedly high levels of insulin from exposing the pancreas to too much fat. Insulin-resistant diabetes can come from eating too much fat as well as from too much sugar.
Almost 50 percent of North Americans will eventually develop diabetes or pre-diabetes, because we eat too much and exercise too little. This is sad because you can prevent and treat most cases of type II diabetes (insulin-resistant diabetes) by getting fat out of your liver and muscles, and increasing the size of your muscles with resistance exercise.
Many people with high blood sugar levels are told by their doctors that they do not have diabetes because their fasting blood sugar levels are below 100 mg/dl, which is considered normal. Early in the disease, diabetics often have a "normal" fasting blood sugar, but one hour after they eat, their blood sugar levels rise above 140, which signals that they are at increased risk for heart attacks, strokes, cancers, nerve damage and premature death.
A low fasting blood sugar or an abnormally low HbA1C (a test of the amount of sugar stuck on cells) may increase risk for heart attacks. Researchers followed almost 5000 people for 13 years and found that having a very low fasting blood sugar (<80 mg/dL) and very low HbA1c (<5.0 percent) is strongly associated with increased risk for heart attacks and premature death.
A study this month shows how people who eat a lot of sugar can develop a liver full of fat that can lead to diabetes. When your blood sugar rises too high, the insulin released by your pancreas converts the sugar to a type of fat called triglycerides. HDL (good) cholesterol then carries the triglycerides to your liver where they are stored to cause a fatty liver.
You can tell if you are at high risk for diabetes if you store fat primarily in your belly. Pinch your belly; if you can pinch an more than an inch of fat under the skin there, you are at increased risk and should get a blood test called HBA1C. Having high blood levels of triglycerides and low levels of the good HDL cholesterol that helps prevent heart attacks also increases your risk for diabetes.
Most people who develop diabetes in later life can be controlled so that they are not at increased risk for the many complications of diabetes such as heart attacks, strokes, blindness, deafness, amputations, kidney failure, burning foot syndrome, venous insufficiency with ulceration and stasis dermatitis.
More than seventy percent of North Americans adults will become diabetic or prediabetic; diseases that are curable with lifestyle changes and not curable by drugs. Insulin insensitivity (failure to respond to insulin) causes the majority of all cases of type II diabetes and prediabetes.
Data from nine studies of 307,099 middle-aged people followed for up to 28 years shows that those who ate lots of plant-based foods and restricted meat had a 23 percent lower risk of developing type 2 diabetes, and those who ate the most vegetables had a 30 percent lower risk of developing diabetes than...
More than 30 million adults in North America suffer from diabetes and another 85 million have pre-diabetes that is likely to become full-blown diabetes within five years. Research papers published this month suggest that people who are diabetic or likely to become diabetic should eat a very large amount of plant fiber and restrict animal products, particularly red meat and processed meats.
A just-published study shows that drinking either sugared or artificially-sweetened drinks is associated with increased risk for diabetes (Am J Clin Nutr, Jun 28, 2017). Of 64,850 post-menopausal women followed for 8.4 years, 4675 developed diabetes.
A review of nine separate trials showed that diabetics who switched to vegetarian diets had significantly lower HbA1c (a measure of cell damage from high blood sugar levels), fasting blood sugar, LDL (bad) cholesterol, body weight and waist circumference. The studies included 664 diabetics who were taking oral sugar-lowering drugs, insulin, cholesterol-lowering drugs and/or blood pressure medications.
Eating mammal meat or processed meats is associated with increased risk for diabetes, particularly if the meat is cooked at high temperatures. The authors showed that eating red or processed meat is associated with excess fat in the liver that can cause high blood sugar levels.
Non-alcoholic fatty liver disease occurs when excess fat is stored in your liver. The most common cause of liver disease used to be alcohol, but now it is far more likely to be caused by eating sugar-added foods and drinking anything with sugar in it. Up to 12 percent of people with fatty livers keep on adding fat to their livers to develop Non-Alcoholic Steato Hepatitis (NASH), which is liver inflammation and permanent liver damage called cirrhosis and can cause liver cancer .
A review of the world's literature shows that high-intensity exercise, particularly interval training, causes greater reduction in HbA1c in diabetics than less intense exercise. HbA1c measures cell damage from high blood sugar levels. Many studies show that increasing exercise intensity controls blood sugar in diabetics more effectively than just increasing the duration of exercise.
Exercising before or after eating helps to protect you from having a high rise in blood sugar after meals. Even light exercise before or after you eat can prevent a high rise in blood sugar and the damage it can cause.
Many doctors are missing a chance to save the lives of their patients by not ordering a blood sugar level one hour after eating a meal. Having a normal fasting blood sugar (<100 mg/dl) does not rule out diabetes.
Very exciting research from Princeton University explains how taking in sugared drinks and any sugar added to foods (not in whole fruits and vegetables) can cause diabetes. The authors of this study show that fructose is converted to glucose primarily in the intestines, and not in the liver as scientists thought previously.
In small studies in animals and humans, vinegar has been shown to reduce the rise in blood sugar and insulin that normally occurs after a meal. However, I cannot find any large studies to show that vinegar is an effective treatment for diabetes.
A liver full of fat can be caused by anything that damages the liver. Doctors used to separate liver damage into that caused by alcohol and that not caused by alcohol (non-alcoholic fatty liver disease or NAFLD). Now we know that a liver damaged by excess alcohol has the same harmful consequences as a...
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